These outcomes show that Ca2+ influx into the axon happened after glutamate excitotoxicity in paranodal myelin is mediated by kainate and NMDA receptors
This result plainly demonstrates that calpain performs an essential function in the pathway of glutamate excitotoxicity.Impact of glutamate (Glut) and 4-AP on CAPs in ex vivo spinal ventral columns. (A)…